Specifically, the antimetamorphic action of Kr-h1 is reciprocally repressed by the expression of E93, which is regulated by ecdysone and its receptor (EcR) and the master trigger of imaginal molt and neoteny (Ureña et al., 2014 Bell es and Santos, 2014 Kayukawa et al., 2017 Chafino et al., 2018 Vea et al., 2019). ![]() Briefly, after JH binds to the receptor Methoprene-tolerant (Met), this complex upregulates the expression of Krüppel-homolog 1 (Kr-h1), which is known as the transducer of the antimetamorphic signal of JH (Minakuchi et al., 2008(Minakuchi et al.,, 2009Charles et al., 2011 Jindra et al., 2021 Kayukawa et al., 2012).
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